MECHANISMS OF CELLULAR CYTOTOXICITY INDUCTION IN GASTRIC MUCOSAL INFLAMMATION
نویسندگان
چکیده
منابع مشابه
Mucosal inflammation in idiopathic bronchiectasis: cellular and molecular mechanisms.
Bronchiectasis is a chronic and debilitating lung disease, characterised by irreversible dilatation of the bronchi as consequence of airway injury and remodelling due to recurrent or chronic airway inflammation and infection. The underlying aetiologies include autoimmune diseases, severe infections, genetic abnormalities and acquired disorders. The pathogenesis of bronchiectasis is poorly under...
متن کاملGASTRIC MUCOSAL PROTECTION Peripheral mechanisms of gastric mucosal protection: mediators, receptors Gastric mucosal barrier to acid
Gastric mucosal integrity can be influenced both by peripheral and central mechanisms. In the last decades the peripheral mechanisms of gastric mucosal defense have been intensively studied, and many details have been clarified. Some of these results have served as a basis for the development of new strategies, therapeutic targets in the treatment of gastric mucosal lesions. On the other hand, ...
متن کاملCellular mechanisms of inflammation
The second meeting of the contact group placed under the patronage of the Fonds National de la Recherche Scientique (FNRS, Belgium) and entitled ‘Cellular Mechanisms of In ammation’ held in Yvoir, Belgium, on 11 October 1996. This meeting began with the master lecture by Dr R. Monteiro (INSERM, Paris) on the potential role of the IgA Fc receptors (FcaR) in the catabolism of IgA. This set the ...
متن کاملMechanisms ofAntibody-dependent Cellular Cytotoxicity
cyte (HRBC) targets in suspension cultures indicative of abnormal intracellular postphagocytic killing. However, when phagocytosis was prevented by using a monolayer of antibody-coated HRBC targets, CGD monocytes, neutrophils, and lymphocytes exhibited normal ADCC activity. Similarly, antibody-coated HRBC targets in suspension could be lysed normally by CGD effector cells when phagocytosis was ...
متن کاملCellular mechanisms of neurogenic inflammation.
Since the initial observations that stimulation of sensory neurons produces vasodilation, plasma extravasation, and hypersensitivity, much progress has been made in understanding the etiology of neurogenic inflammation. Studies have focused largely on the role of the neuropeptides, substance P and calcitonin gene-related peptide, which are released in the periphery by activation of small diamet...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Medical Immunology (Russia)
سال: 2017
ISSN: 2313-741X,1563-0625
DOI: 10.15789/1563-0625-2017-6-673-682